Saturday, 20 Apr 2024

First in the world, a pill that will stops you to getting fat even on an unhealthy diet

news24xx


IllustrationIllustration

News24xx.com -  For fat woman, don't worry, now your obesity could be stopped with a world-first pill which can to stops the body producing fat cells. A quarter of people around the world are expected to be obese by 2045.

A study has claimed the scientists have invented a drug which stops the fat-creating process in the body, which they say could help tackle heart disease, cancer and dementia. The researchers have hailed their findings a 'major step forward' in tackling the crisis.

In a study on mice, the animals given the drug managed to stay slim even when they ate high-fat foods.

The pill – named PO53 – causes fats like cholesterol to be burned up by muscles instead of building up in the body, and experts hope it will also work in humans.

Scientists at the University of New South Wales in Sydney, Australia, began their experiment to try and find a way to prevent type 2 diabetes.

The condition is triggered by obesity, but is caused by resistance to the blood-sugar controlling hormone insulin.

But instead of affecting blood sugar, the drug created by the scientists stopped the mice's bodies developing body fat.

Researchers say their discovery is the first time scientists have managed to stop this process. 

Reducing obesity could cut cancer and heart disease deaths 

The pill could have much bigger implications and cutting down obesity rates could reduce people's risk of getting type 2 diabetes, cancer, heart disease and dementia, all of which are more likely to affect fat people.

The drug works by blocking an enzyme called ceramide synthase 1 (CerS1). And the drug makes body burn fat instead of storing it 

But PO53 stops the activity of fat-producing enzyme CerS1 in its tracks, reducing fat across the whole body without effecting insulin resistance in mice fed on a high-fat diet.
 
 
 

 

 

 

NEWS24XX.COM/DEV/RED 





loading...
Versi Mobile
Most Popular
Loading...